Browsing by Autor "Vidal Felices"

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Detección de una cepa de influenza A (H1N1) pdm09 resistente al Oseltamivir en Perú
(National Institute of Health of Peru, 2014) Josefina García; Vidal Felices; Jorge Gómez; Elizabeth Gómez; V. Alberto Laguna-Torres
Genetic Characterization of Venezuelan Equine Encephalitis Virus from Bolivia, Ecuador and Peru: Identification of a New Subtype ID Lineage
(Public Library of Science, 2009) Patricia V. Aguilar; Alexandra Adams; Víctor Suárez; Luis Beingolea; Jorge Vargas; Stephen R. Manock; Juan Freire; Willan R. Espinoza; Vidal Felices; Ana María Díaz
Venezuelan equine encephalitis virus (VEEV) has been responsible for hundreds of thousands of human and equine cases of severe disease in the Americas. A passive surveillance study was conducted in Peru, Bolivia and Ecuador to determine the arboviral etiology of febrile illness. Patients with suspected viral-associated, acute, undifferentiated febrile illness of <7 days duration were enrolled in the study and blood samples were obtained from each patient and assayed by virus isolation. Demographic and clinical information from each patient was also obtained at the time of voluntary enrollment. In 2005-2007, cases of Venezuelan equine encephalitis (VEE) were diagnosed for the first time in residents of Bolivia; the patients did not report traveling, suggesting endemic circulation of VEEV in Bolivia. In 2001 and 2003, VEE cases were also identified in Ecuador. Since 1993, VEEV has been continuously isolated from patients in Loreto, Peru, and more recently (2005), in Madre de Dios, Peru. We performed phylogenetic analyses with VEEV from Bolivia, Ecuador and Peru and compared their relationships to strains from other parts of South America. We found that VEEV subtype ID Panama/Peru genotype is the predominant one circulating in Peru. We also demonstrated that VEEV subtype ID strains circulating in Ecuador belong to the Colombia/Venezuela genotype and VEEV from Madre de Dios, Peru and Cochabamba, Bolivia belong to a new ID genotype. In summary, we identified a new major lineage of enzootic VEEV subtype ID, information that could aid in the understanding of the emergence and evolution of VEEV in South America.
Hantavirus Pulmonary Syndrome in Santa Cruz, Bolivia: Outbreak Investigation and Antibody Prevalence Study
(Public Library of Science, 2012) Joel M. Montgomery; Patrick J. Blair; Darin S. Carroll; James N. Mills; Alberto Gianella; Naomi Iihoshi; Ana M. Briggiler; Vidal Felices; Milagros Salazar; James G. Olson
We report the results of an investigation of a small outbreak of hantavirus pulmonary syndrome in 2002 in the Department of Santa Cruz, Bolivia, where the disease had not previously been reported. Two cases were initially reported. The first case was a physician infected with Laguna Negra virus during a weekend visit to his ranch. Four other persons living on the ranch were IgM antibody-positive, two of whom were symptomatic for mild hantavirus pulmonary syndrome. The second case was a migrant sugarcane worker. Although no sample remained to determine the specific infecting hantavirus, a virus 90% homologous with Río Mamoré virus was previously found in small-eared pygmy rice rats (Oligoryzomys microtis) trapped in the area. An antibody prevalence study conducted in the region as part of the outbreak investigation showed 45 (9.1%) of 494 persons to be IgG positive, illustrating that hantavirus infection is common in Santa Cruz Department. Precipitation in the months preceding the outbreak was particularly heavy in comparison to other years, suggesting a possible climatic or ecological influence on rodent populations and risk of hantavirus transmission to humans. Hantavirus infection appears to be common in the Santa Cruz Department, but more comprehensive surveillance and field studies are needed to fully understand the epidemiology and risk to humans.
Molecular epidemiology of dengue virus type 3 in Northern South America: 2000–2005
(Elsevier BV, 2008) Tadeusz J. Kochel; Patricia Aguilar; Vidal Felices; Guillermo Comach; Cristhopher D. Cruz; Aracely Alava; Jorge Vargas; James G. Olson; Patrick J. Blair
Reemergence of Bolivian Hemorrhagic Fever, 2007–2008
(Centers for Disease Control and Prevention, 2009) Patricia V. Aguilar; Wilfredo Camargo; Jorge Vargas; Carolina Guevara; Yelin Roca; Vidal Felices; V. Alberto Laguna-Torres; Robert B. Tesh; Thomas G. Ksiazek; Tadeusz J. Kochel
age.In particular, suspected childhood TB patients without an identifiable TB contact and with normal immune status were subjected to further investigations.Multidisciplinary management, including enhanced laboratory diagnosis of atypical bony lesions in infants and children, is recommended for any suspected TB infection.Once BCGrelated infection is confirmed, medical treatment has to be consistent.