JM-20 administration to animals with lesion of the nigrostriatal dopamine pathway induced by 6-hydroxydopamine, partially reverses motor damage and oxidative stress

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dc.contributor.authorLuis Arturo Fonseca-Fonseca
dc.contributor.authorLaura Reina Taño Portuondo
dc.contributor.authorJeney Ramírez-Sánchez
dc.contributor.authorNancy Pavón Fuentes
dc.contributor.authorAbel Mondelo Rodríguez
dc.contributor.authorVíctor Diógenes Amaral da Silva
dc.contributor.authorSílvia Lima Costa
dc.contributor.authorYanier Núñez-Figueredo
dc.coverage.spatialBolivia
dc.date.accessioned2026-03-22T19:35:35Z
dc.date.available2026-03-22T19:35:35Z
dc.date.issued2025
dc.description.abstractOur study provides the preclinical evidence to support the long-term neuroprotective potential of JM-20 in 6-OHDA hemiparkinson rat model, pointing out to its possible use as a disease-modifying agent in PD.
dc.identifier.doi10.1080/01616412.2025.2490089
dc.identifier.urihttps://doi.org/10.1080/01616412.2025.2490089
dc.identifier.urihttps://andeanlibrary.org/handle/123456789/76959
dc.language.isoen
dc.publisherTaylor & Francis
dc.relation.ispartofNeurological Research
dc.sourceCentro de Investigación y Desarrollo
dc.subjectPars compacta
dc.subjectSubstantia nigra
dc.subjectDopamine
dc.subjectNeuroprotection
dc.subjectHydroxydopamine
dc.subjectStriatum
dc.subjectOxidative stress
dc.subjectOxidopamine
dc.subjectLesion
dc.subjectParkinson's disease
dc.titleJM-20 administration to animals with lesion of the nigrostriatal dopamine pathway induced by 6-hydroxydopamine, partially reverses motor damage and oxidative stress
dc.typearticle

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