[Isoniazid acetylation, its relation to genetic and environmental altitude factors].

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dc.contributor.authorGérald Lopez
dc.contributor.authorM L Navarro de Claure
dc.coverage.spatialBolivia
dc.date.accessioned2026-03-22T17:30:12Z
dc.date.available2026-03-22T17:30:12Z
dc.date.issued1987
dc.description.abstractSince the discovery of isoniacid in 1952, it has become apparent that there are considerable variations in the manner in which individuals react to the drug. We studied 90 patients from La Paz (3.600 m. over sea level) and 50 patients from Santa Cruz (470 m. over sea level). Our results in the two groups tested suggest that the high altitude hypoxia increases the velocity of inactivation. Despite to the influence of hypoxia it was seen that both groups have the some frequency of rapid and slow inactivators.
dc.identifier.urihttps://pubmed.ncbi.nlm.nih.gov/8929085
dc.identifier.urihttps://andeanlibrary.org/handle/123456789/64555
dc.language.isoen
dc.publisherNational Institutes of Health
dc.relation.ispartofPubMed
dc.sourceUniversidad Mayor de San Andrés
dc.subjectHypoxia (environmental)
dc.subjectAcetylation
dc.subjectAltitude (triangle)
dc.subjectIsoniazid
dc.subjectEffects of high altitude on humans
dc.subjectSea level
dc.subjectBiology
dc.title[Isoniazid acetylation, its relation to genetic and environmental altitude factors].
dc.typearticle

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